Search results for "leptin receptor"

showing 10 items of 43 documents

Association between polymorphisms in the leptin promoter and leptin receptor genes and cancer in a mediterranean population of the PREDIMED study

2016

Geneticseducation.field_of_studymedicine.medical_specialtyLeptin receptorbusiness.industryLeptinPopulationCancerHematologymedicine.diseasePredimedEndocrinologyOncologyInternal medicinemedicineeducationbusinessGeneAnnals of Oncology
researchProduct

Leptin/HER2 crosstalk in breast cancer: in vitro study and preliminary in vivo analysis.

2008

Abstract Background Obesity in postmenopausal women is associated with increased breast cancer risk, development of more aggressive tumors and resistance to certain anti-breast cancer treatments. Some of these effects might be mediated by obesity hormone leptin, acting independently or modulating other signaling pathways. Here we focused on the link between leptin and HER2. We tested if HER2 and the leptin receptor (ObR) can be coexpressed in breast cancer cell models, whether these two receptors can physically interact, and whether leptin can transactivate HER2. Next, we studied if leptin/ObR can coexist with HER2 in breast cancer tissues, and if presence of these two systems correlates wi…

LeptinTranscriptional Activationmedicine.medical_specialtyCancer ResearchReceptor ErbB-2Breast Neoplasmslcsh:RC254-282Breast cancerSurgical oncologyRisk FactorsInternal medicineCell Line TumormedicineGeneticsHumansObesityReceptorskin and connective tissue diseasesneoplasmsLeptin receptorbusiness.industryLeptinCarcinoma Ductal BreastReceptor Cross-Talklcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseImmunohistochemistryGene Expression Regulation NeoplasticPostmenopauseEndocrinologyOncologyImmunohistochemistryReceptors LeptinFemaleSignal transductionbusinessImmunostainingProtein BindingResearch ArticleBMC cancer
researchProduct

Dose-dependent biphasic leptin-induced proliferation is caused by non-specific IL-6/NF-κB pathway activation in human myometrial cells

2015

Background and Purpose Leptin, an adipokine synthesized by the placenta during pregnancy, has been proposed for the management of preterm labour (PTL), as it is able to prevent in vitro uterine contractility and remodelling associated with labour onset. Another common feature of labour onset is the phenotypic switch of myometrial smooth muscle cells from a proliferative to a hypertrophic state. As proliferative effects have been demonstrated for leptin in other tissues, we aimed to investigate its ability to induce myometrial proliferation and thus to maintain uterine quiescence. Experimental Approach We stimulated human primary myometrial smooth muscle cells with leptin in the presence or …

Pharmacologymedicine.medical_specialtyLeptin receptorLeptindigestive oral and skin physiologyMyometriumAdipokineStimulationBiologyEndocrinologyInternal medicinemedicineMyocyteSignal transductionReceptorBritish Journal of Pharmacology
researchProduct

Notch-1 decreased expression contributes to leptin receptor downregulation in nasal epithelium from allergic turbinates

2019

Abstract BACKGROUND: Allergic rhinitis is characterized by a remodeling of nasal epithelium. Since the Notch and TGF-β signaling pathways are known to be involved in cell differentiation and remodeling processes and leptin adipokine has already been identified as a marker for homeostasis in human bronchial and nasal epithelial cells of asthmatics, roles played by these pathways have been investigated for chronic allergic rhinitis. METHODS: The leptin/leptin receptor expression has been investigated in a study with 40 biopsies from allergic (AR, n = 18) and non-allergic (C, n = 22) inferior turbinates, using immunohistochemistry, immunofluorescence staining and RT-PCR. In addition, extracts …

0301 basic medicineAdultLeptinMalemedicine.medical_specialtyBiopsyPrimary Cell CultureAdipokineTurbinatesCell LineTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineDownregulation and upregulationInternal medicinemedicineHomeostasisHumansRNA MessengerReceptor Notch1610 Medicine & healthReceptorMolecular BiologyNotch 1Leptin receptorChemistryLeptindigestive oral and skin physiologyEpithelial CellsMiddle AgedRhinitis AllergicAllergic rhinitis Epithelium Leptin NotchEpitheliumNasal Mucosa030104 developmental biologyEndocrinologymedicine.anatomical_structureGene Expression Regulation030220 oncology & carcinogenesisCase-Control StudiesMolecular MedicineReceptors LeptinFemaleSignal transductionSignal Transduction
researchProduct

Functional analysis of the -2548G/A leptin gene polymorphism in breast cancer cells

2009

Leptin is overexpressed in human breast tumors and is produced by breast cancer cells in response to obesity-related stimuli. The leptin promoter polymorphism Lep-2548G/A can be associated with increased leptin secretion by adipocytes and elevated cancer risk. However, molecular mechanisms underlying the link between Lep-2548G/A and breast cancer have never been addressed. Lep-2548G/A is proximal to a binding site for the transcriptional factor Sp1. Furthermore nucleolin, a transcriptional repressor, can bind Sp1 or its consensus site. Consequently, we focused on the impact of Lep-2548G/A on Sp1- and nucleolin-dependent leptin transcription in breast cancer cells. The Lep-2548G/A was identi…

LeptinChromatin ImmunoprecipitationCancer Researchmedicine.medical_specialtyGenotypeSp1 Transcription FactorBlotting WesterneducationAdipokineBreast NeoplasmsBiologyBody Mass IndexBreast cancerInternal medicineTumor Cells CulturedmedicineHumansHypoglycemic AgentsInsulinObesityRNA MessengerPromoter Regions Genetichealth care economics and organizationsPolymorphism GeneticLeptin receptorReverse Transcriptase Polymerase Chain ReactionLeptinRNA-Binding ProteinsCancerPhosphoproteinsmedicine.diseaseEndocrinologyOncologyCancer researchImmunohistochemistryBreast diseaseNucleolinhormones hormone substitutes and hormone antagonistsInternational Journal of Cancer
researchProduct

Dietary xenoestrogens differentially impair 3T3-L1 preadipocyte differentiation and persistently affect leptin synthesis

2008

International audience; Recent observations have highlighted adipogenesis alterations under exposure to several xenoestrogens at critical stages, and pointed at their possible involvement in the pathogenesis of obesity. However, it remains unclear whether these effects are mediated by classical estrogen receptor (ER) binding and subsequent transcriptional modulation. The aim of this study was to determine the (anti-)adipogenic impact of apigenin, bisphenol A, genistein and 17β-estradiol at the onset of adipose cell maturation, and to correlate it to their estrogenic potential. In steroid-free conditions, 3T3-L1 preadipocytes were induced to differentiate in the presence of xenoestrogens for…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryGene ExpressionAdipose tissueEstrogen receptorBiochemistryMicechemistry.chemical_compound0302 clinical medicineEndocrinologyAdipocyte[SDV.IDA]Life Sciences [q-bio]/Food engineeringAdipocytesApigeninESTROGEN RECEPTORS0303 health sciencesEstradiolReverse Transcriptase Polymerase Chain ReactionLeptinCell Differentiation[SDV.IDA] Life Sciences [q-bio]/Food engineeringGenisteinReceptors EstrogenAdipogenesis030220 oncology & carcinogenesisIntercellular Signaling Peptides and ProteinsMolecular Medicinehormones hormone substitutes and hormone antagonistsmedicine.medical_specialty[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringXENOESTROGENSEnzyme-Linked Immunosorbent AssayBiologyModels Biological03 medical and health sciencesLEPTINPhenols3T3-L1 CellsInternal medicinemedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRNA Messengerfas ReceptorBenzhydryl CompoundsMolecular Biology030304 developmental biology3T3-L1Leptin receptorCalcium-Binding ProteinsEstrogens3T3-L1Cell BiologyADIPOGENESISPPAR gammaSteroid hormoneEndocrinologychemistry
researchProduct

Resistin, visfatin, leptin and omentin are differently related to hormonal and metabolic parameters in growth hormone-deficient children

2016

PURPOSE: The effect of growth hormone (GH) on adipose tissue and the role of adipokines in modulating metabolism are documented, but with discordant data. Our aim was to evaluate the impact of GH treatment on a series of selected adipokines known to have a metabolic role and poorly investigated in this setting. METHODS: This is a prospective study. Thirty-one prepubertal children (25 M, 6 F; aged 8.5 ± 1.6 years) with isolated GH deficiency treated with GH for at least 12 months and 30 matched controls were evaluated. Auxological and metabolic parameters, insulin sensitivity indexes, leptin, soluble leptin receptor, adiponectin, visfatin, resistin, omentin, adipocyte fatty acid-binding prot…

0301 basic medicineLeptinMalemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentAdipokineAdipose tissue030209 endocrinology & metabolismEnzyme-Linked Immunosorbent AssayGPI-Linked ProteinsAdipokines; Children; Growth hormone; Insulin sensitivitySettore MED/13 - Endocrinologia03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInternal medicineAdipocyteLectinsAdipokinemedicineHumansResistinProspective StudiesChildNicotinamide PhosphoribosyltransferaseChildrenGrowth hormoneGrowth DisordersLeptin receptorAdiponectinbusiness.industryHuman Growth HormoneLeptinInsulinInsulin sensitivityHormones030104 developmental biologyEndocrinologychemistryCase-Control StudiesChild PreschoolCytokinesResistinFemalebusinessBiomarkers
researchProduct

Serum visfatin levels in acromegaly: Correlation with disease activity and metabolic alterations

2015

Objective: The studies that have extensively evaluated the relation between adipokines and metabolic parameters in acromegaly treatment are quite discordant. We aimed to evaluate and correlate a set of selected adipokines, known to have a metabolic role, with the disease activity, metabolic status and treatment modalities. Design: Data of 56 consecutive acromegalic patients (31 M and 25 F; aged 54 ± 12 years), admitted to the section of Endocrinology of the University of Palermo during the years 2005-2014, including 16 newly diagnosed untreated (ND), 21 during therapy with somatostatin analogues (SA), 12 with pegvisomant (PE) and 7 after surgical treatment (SU), grouped into uncontrolled (g…

AdultLeptinMalemedicine.medical_specialtyEndocrinology Diabetes and MetabolismAdipokineFatty Acids NonesterifiedSettore MED/13 - EndocrinologiaInsulin resistanceEndocrinologyAcromegaly; Adipokines; Growth hormone; Endocrinology; Endocrinology Diabetes and MetabolismAdipokinesInternal medicineAdipokineAcromegalyMedicineHumansNicotinamide PhosphoribosyltransferaseGrowth hormoneTriglyceridesAdiposityAgedLeptin receptorAdiponectinbusiness.industryLeptinCholesterol HDLMiddle Agedmedicine.diseaseEndocrinologyCross-Sectional StudiesPegvisomantAcromegalyCytokinesResistinFemaleInsulin ResistancebusinessBiomarkersmedicine.drug
researchProduct

The role of the leptin in reproduction.

2006

Since its discovery in 1994, leptin has appeared to be a pleiotrophic hormone, governing energy homeostasis and affecting many tissues in the body. Numerous pieces of evidence have accumulated showing that leptin potentially plays an important role in the control of the reproductive function.This review presents the major concepts for the role of leptin in the modulation of reproductive function. As a marker of the nutritional status, leptin affects the hypothalamo-pituitary-gonadal axis, regulating gonadotrophin-releasing hormone and luteinising hormone secretion, and appears to be a permissive factor in the onset of the puberty. This protein and its receptor have been found in the reprodu…

Leptinmedicine.medical_specialtyLeptin receptorLeptinmedia_common.quotation_subjectReproductiondigestive oral and skin physiologyOvaryObstetrics and GynecologyBiologyEnergy homeostasisEndometriumEndocrinologyPregnancyInternal medicinemedicineHumansFemaleEmbryo ImplantationReproductionhormones hormone substitutes and hormone antagonistsHormonemedia_commonCurrent opinion in obstetricsgynecology
researchProduct

Leptin and Its Receptor are Overexpressed in Brain Tumors and Correlate with the Degree of Malignancy

2009

Although leptin and its receptor (ObR) have emerged as important cancer biomarkers, the role of the leptin system in brain tumor development remains unknown. We screened 87 human brain tumor biopsies using immunohistochemistry and detected leptin and ObR in 55.2% and 60.9% cases, respectively. In contrast, leptin and ObR were absent in 14 samples of normal brain tissue. The presence of leptin correlated with ObR with overall concordance 80.5%. The leptin/ObR system was highly expressed in glioblastomas and anaplastic astrocytomas, while lower expression of both markers was noted in low-grade astrocytomas and gangliogliomas. The association between leptin/ObR and the degree of tumor malignan…

STAT3 Transcription Factornovel biomarkermedicine.medical_specialtyBlotting WesternBrain tumorFluorescent Antibody TechniqueCell CountBiologyleptinArticlePathology and Forensic MedicineCell Line TumorInternal medicinemedicineHumansleptin receptorProtein kinase BCell ProliferationLeptin receptorBrain NeoplasmsGeneral NeuroscienceLeptindigestive oral and skin physiologyglioblastomaBrainGliomamalignant progressionCell cyclemedicine.diseaseImmunohistochemistryOncogene Protein v-aktglioblastoma; leptin; leptin receptor; malignant progression; novel biomarkerKi-67 AntigenEndocrinologyTumor progressionReceptors LeptinImmunohistochemistryCancer biomarkersNeurology (clinical)hormones hormone substitutes and hormone antagonistsSignal TransductionBrain Pathology
researchProduct